Novel phage peptides attenuate beta amyloid-42 catalysed hydrogen peroxide production and associated neurotoxicity

Taddei, K.; Laws, S.M.; Verdile, G.; Munns, S.; D'Costa, K.; Harvey, A.R.; Martins, I.J.; Hill, F.; Levy, E.; Shaw, John; Martins, Ralph

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Novel phage peptides attenuate beta amyloid-42 catalysed hydrogen peroxide production and associated neurotoxicity

Date

2010

Authors

Taddei, K.

Laws, S.M.

Verdile, G.

Munns, S.

D'Costa, K.

Harvey, A.R.

Martins, I.J.

Hill, F.

Levy, E.

Shaw, John

Publisher

Elsevier

Abstract

Amyloid-β (Aβ) peptides play a central role in the pathogenesis of Alzheimer's disease. There is accumulating evidence that supports the notion that the toxicity associated with human Aβ (both 40 and 42) is dependent on its superoxide dismutase (SOD)-l

URI

http://hdl.handle.net/1885/60954

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Open Access Research

Source

Neurobiology of Aging

Type

Journal article

DOI

10.1016/j.neurobiolaging.2008.03.023

Restricted until

2037-12-31

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Novel phage peptides attenuate beta amyloid-42 catalysed hydrogen peroxide production and associated neurotoxicity

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